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ANP (1-28); Carperitide,Catalog Number: KS062018,CAS NO.: 89213-87-6 Atrial natriuretic peptide (ANP) is an endogenous peptide generated by proteolysis of prepro-ANP that is secreted by cardiomyocytes in the heart. It has effects on the renal and cardiovascular systems that decrease vasoconstriction, inhibit renin secretion, and increase sodium excretion. Human ANP binds to ANP receptors on cultured vascular smooth muscle cells (VSMCs) with a Kd value of approximately 1-2 nM

ANP (1-28); Carperitide,Catalog Number: KS063038,CAS NO.: 89213-87-6 Atrial natriuretic peptide (ANP) is an endogenous peptide generated by proteolysis of prepro-ANP that is secreted by cardiomyocytes in the heart. It has effects on the renal and cardiovascular systems that decrease vasoconstriction, inhibit renin secretion, and increase sodium excretion. Human ANP binds to ANP receptors on cultured vascular smooth muscle cells (VSMCs) with a Kd value of approximately 1-2 nM

CNP-53 (Human),CAS#:141294-77-1,Catalog Number:KS181022 C-type natriuretic peptide (CNP) is produced by the vascular endothelium and acts predominantly in an autocrine/paracrine fashion. It has vasodilative properties and is likely significant in regulating vascular tone, local blood flow and systemic blood pressure. Like atrial natriuretic peptide (ANP), CNP plays a regulatory role in pancreatic and other digestive secretions. CNP is an important regulator of cartilage

Atrial natriuretic peptide (ANP) is an endogenous peptide generated by proteolysis of prepro-ANP that is secreted by cardiomyocytes in the heart. It has effects on the renal and cardiovascular systems that decrease vasoconstriction, inhibit renin secretion, and increase sodium excretion. Human ANP binds to ANP receptors on cultured vascular smooth muscle cells (VSMCs) with a Kd value of approximately 1-2 nM and increases cGMP levels in a dose-dependent manner. It relaxes